Browsing Research Publications by Author "Wan, J."

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Genome-wide DNA hypermethylation opposes healing in patients with chronic wounds by impairing epithelialmesenchymal transition

Singh, K.; Roy, S.; Sen, C. K.; Rustag, Y.; Abouhashem, A.S.; Tabasum, S.; Verma, P.; Hernandez, E.; Khona, D.K.; Pal, D.; Mohanty, S.K.; Kumar, M.; Srivastava, R.; Guda, P.R.; Verma, S.S.; Mahajan, S.; Killian, J.A.; Walker, L. A.; Ghatak, S.; Mathew-Steiner, S.S.; Wanczyk, K.E.; Liu, S.; Wan, J.; Yan, P.; Bundschuh, R.; Khanna, G. M.; Murphy, M.P. (2022-10-27)

An extreme chronic wound tissue microenvironment causes epigenetic gene silencing. An unbiased whole-genome methylome was studied in the wound-edge tissue of patients with chronic wounds. A total of 4,689 differentially ...
Identification of a physiologic vasculogenic fibroblast state to achieve tissue repair

Pal, D.; Ghatak, S.; Singh, K.; Abouhashem, A.S.; Kumar, M.; S El Masry, M.; Mohanty, S.K.; Palakurti, R.; Rustagi, Y.; Tabasum, S.; Khona, D.K.; Khanna, S.; Kacar, S.; Srivastava, R.; Bhasme, P.; Verma, S.S.; Hernandez, E.; Sharma, A.; Reese, D.; Verma, P.; Ghosh, N.; Gorain, M.; Wan, J.; Liu, S.; Sen, C.K. (2024-05-11)

Tissue injury to skin diminishes miR-200b in dermal fibroblasts. Fibroblasts are widely reported to directly reprogram into endothelial-like cells and we hypothesized that miR-200b inhibition may cause such changes. We ...

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